Investigación de la Infección
Harvard Medical School
Boston, Estados UnidosPublicaciones en colaboración con investigadores/as de Harvard Medical School (7)
2012
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The translational repressor T-cell intracellular antigen-1 (TIA-1) is a key modulator of Th2 and Th17 responses driving pulmonary inflammation induced by exposure to house dust mite
Immunology Letters, Vol. 146, Núm. 1-2, pp. 8-14
2010
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Fas-activated serine/threonine phosphoprotein promotes immune-mediated pulmonary inflammation
Journal of Immunology, Vol. 184, Núm. 9, pp. 5325-5332
2006
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Identification and characterization of two related murine genes, Eat2a and Eat2b, encoding single SH2-domain adapters
Immunogenetics, Vol. 58, Núm. 1, pp. 15-25
2004
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SAP increases FynT kinase activity and is required for phosphorylation of SLAM and Ly9
International Immunology, Vol. 16, Núm. 5, pp. 727-736
2002
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Molecular dissection of the signaling and costimulatory functions of CD150 (SLAM): CD150/SAP binding and CD150-mediated costimulation
Blood, Vol. 99, Núm. 3, pp. 957-965
2001
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Cell surface receptors Ly-9 and CD84 recruit the X-linked lymphoproliferative disease gene product SAP
Blood, Vol. 97, Núm. 12, pp. 3867-3874
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Characterization of SH2D1A Missense Mutations Identified in X-linked Lymphoproliferative Disease Patients
Journal of Biological Chemistry, Vol. 276, Núm. 39, pp. 36809-36816