Edición Génica para el Estudio de Canales Iónicos Vasculares y Proteínas Mitocondriales
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Harvard Medical School
Boston, Estados UnidosHarvard Medical School-ko ikertzaileekin lankidetzan egindako argitalpenak (10)
2016
2014
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NAD + protects against EAE by regulating CD4 + T-cell differentiation
Nature Communications, Vol. 5
2012
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B cell-intrinsic deficiency of the Wiskott-Aldrich syndrome protein (WASp) causes severe abnormalities of the peripheral B-cell compartment in mice
Blood, Vol. 119, Núm. 12, pp. 2819-2828
2010
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Fas-activated serine/threonine phosphoprotein promotes immune-mediated pulmonary inflammation
Journal of Immunology, Vol. 184, Núm. 9, pp. 5325-5332
2008
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WASP confers selective advantage for specific hematopoietic cell populations and serves a unique role in marginal zone B-cell homeostasis and function
Blood, Vol. 112, Núm. 10, pp. 4139-4147
2007
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Transcellular Diapedesis Is Initiated by Invasive Podosomes
Immunity, Vol. 26, Núm. 6, pp. 784-797
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WIP is a chaperone for Wiskott-Aldrich syndrome protein (WASP)
Proceedings of the National Academy of Sciences of the United States of America, Vol. 104, Núm. 3, pp. 926-931
2006
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WIP and WASP play complementary roles in T cell homing and chemotaxis to SDF-1α
International Immunology, Vol. 18, Núm. 2, pp. 221-232
2005
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Defective nuclear translocation of nuclear factor of activated T cells and extracellular signal-regulated kinase underlies deficient IL-2 gene expression in Wiskott-Aldrich syndrome
Journal of Allergy and Clinical Immunology, Vol. 116, Núm. 6, pp. 1364-1371
2004
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WIP Regulates Signaling via the High Affinity Receptor for Immunoglobulin E in Mast Cells
Journal of Experimental Medicine, Vol. 199, Núm. 3, pp. 357-368