Publicaciones (31) Publicaciones de YOLANDA BAYON PRIETO

2024

  1. The IRE1α-XBP1 arm of the unfolded protein response is a host factor activated in SARS-CoV-2 infection

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1870, Núm. 5

2022

  1. PSTPIP1-LYP phosphatase interaction: structural basis and implications for autoinflammatory disorders

    Cellular and Molecular Life Sciences, Vol. 79, Núm. 2

2021

  1. Fisiología del sistema endocrino: manual del estudiante

    Ediciones Universidad de Valladolid (EdUVa)

2016

  1. The extended family of protein tyrosine phosphatases

    Methods in Molecular Biology (Humana Press Inc.), pp. 1-23

2014

  1. Proline-serine-threonine phosphatase interacting protein 1 inhibition of T-cell receptor signaling depends on its SH3 domain

    FEBS Journal, Vol. 281, Núm. 17, pp. 3844-3854

2013

  1. The Autoimmunity Risk Variant LYP-W620 Cooperates with CSK in the Regulation of TCR Signaling

    PLoS ONE, Vol. 8, Núm. 1

2009

  1. Characterization of new substrates targeted by Yersinia tyrosine phosphatase YopH

    PLoS ONE, Vol. 4, Núm. 2

2008

  1. KCTD5, a putative substrate adaptor for cullin3 ubiquitin ligases

    FEBS Journal, Vol. 275, Núm. 15, pp. 3900-3910

2006

  1. Coupling of C3bi to IgG inhibits the tyrosine phosphorylation signaling cascade downstream Syk and reduces cytokine induction in monocytes

    Journal of Leukocyte Biology, Vol. 79, Núm. 5, pp. 1073-1082

2004

  1. The retinoid antagonist MX781 induces clusterin expression in prostate cancer cells via heat shock factor-1 and activator protein-1 transcription factors

    Cancer Research, Vol. 64, Núm. 16, pp. 5905-5912

2003

  1. Inhibition of IκB kinase by a new class of retinoid-related anticancer agents that induce apoptosis

    Molecular and Cellular Biology, Vol. 23, Núm. 3, pp. 1061-1074

  2. Reduced concentrations of serum enhance the antiproliferative activity of retinoid-related molecules and accelerate the onset of apoptosis

    Biochemical Pharmacology, Vol. 65, Núm. 12, pp. 2021-2030

  3. Z-FA-fmk inhibits effector caspases but not initiator caspases 8 and 10, and demonstrates that novel anticancer retinoid-related molecules induce apoptosis via the intrinsic pathway

    Molecular Cancer Therapeutics, Vol. 2, Núm. 3, pp. 255-263

2002

  1. Retinoids in combination therapies for the treatment of cancer: Mechanisms and perspectives

    Drug Resistance Updates

2001

  1. Brucella lipopolysaccharides induce cyclooxygenase-2 expression in monocytic cells

    Biochemical and Biophysical Research Communications, Vol. 289, Núm. 2, pp. 372-375

  2. Retinoid-related molecules induce cytochrome c release and apoptosis through activation of c-Jun NH2-terminal kinase/p38 mitogen-activated protein kinases

    Cancer Research, Vol. 61, Núm. 23, pp. 8504-8512

2000

  1. Lipopolysaccharides of Brucella abortus and Brucella melitensis induce nitric oxide synthesis in rat peritoneal macrophages

    Infection and Immunity, Vol. 68, Núm. 3, pp. 1740-1745

  2. Stimulation of FcγR receptors induces monocyte chemoattractant protein-1 in the human monocytic cell line THP-1 by a mechanism involving IκB-α degradation and formation of p50/p65 NF-κB/Rel complexes

    International Immunology, Vol. 12, Núm. 4, pp. 547-554

1999

  1. 4-Trifluoromethyl derivatives of salicylate, triflusal and its main metabolite 2-hydroxy-4-trifluoromethylbenzoic acid, are potent inhibitors of nuclear factor κB activation

    British Journal of Pharmacology, Vol. 126, Núm. 6, pp. 1359-1366

  2. Inhibition of cyclooxygenase-2 expression by 4-trifluoromethyl derivatives of salicylate, triflusal, and its deacetylated metabolite, 2- hydroxy-4-trifluoromethylbenzoic acid

    Molecular Pharmacology, Vol. 55, Núm. 4, pp. 753-761