FAC MEDICINA
Centre
Nestlé Health Science
Lausanne, SuizaPublications en collaboration avec des chercheurs de Nestlé Health Science (25)
2024
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Hybrid Evaluation of a Lifestyle Change Program to Prevent the Development of Type 2 Diabetes Among Individuals With Prediabetes: Intended and Observed Changes in Intervening Mechanisms
Journal of primary care & community health, Vol. 15
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The OPTIFAST total and partial meal replacement programme reduces cardiometabolic risk in adults with obesity: Secondary and exploratory analysis of the OPTIWIN study
Diabetes, Obesity and Metabolism, Vol. 26, Núm. 3, pp. 950-960
2023
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Effects of an oral biodegradable device used for 12 weeks on weight reduction, cardiovascular risk factors, satiety, snacking, and meal size
Obesity Pillars, Vol. 8
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SLC25A46 promotes mitochondrial fission and mediates resistance to lipotoxic stress in INS-1E insulin-secreting cells
Journal of Cell Science, Vol. 136, Núm. 8
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Socio-economic factors, mood, primary care utilization, and quality of life as predictors of intervention cessation and chronic stress in a type 2 diabetes prevention intervention (PREVIEW Study)
BMC Public Health, Vol. 23, Núm. 1
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The mitochondrial calcium uniporter (MCU) activates mitochondrial respiration and enhances mobility by regulating mitochondrial redox state
Redox Biology, Vol. 64
2022
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Forming New Health Behavior Habits During Weight Loss Maintenance—The PREVIEW Study
Health Psychology, Vol. 41, Núm. 8, pp. 549-558
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Mitochondrial respiratory chain dysfunction alters ER sterol sensing and mevalonate pathway activity
Journal of Biological Chemistry, Vol. 298, Núm. 3
2021
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Interaction of Diet/Lifestyle Intervention and TCF7L2 Genotype on Glycemic Control and Adiposity among Overweight or Obese Adults: Big Data from Seven Randomized Controlled Trials Worldwide
Health Data Science, Vol. 2021
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Mitochondrial ion channels in pancreatic β-cells: Novel pharmacological targets for the treatment of Type 2 diabetes
British Journal of Pharmacology, Vol. 178, Núm. 10, pp. 2077-2095
2020
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Augmented mitochondrial energy metabolism is an early response to chronic glucose stress in human pancreatic beta cells
Diabetologia, Vol. 63, Núm. 12, pp. 2628-2640
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Protein Lysine Acetylation: Grease or Sand in the Gears of β-Cell Mitochondria?
Journal of Molecular Biology, Vol. 432, Núm. 5, pp. 1446-1460
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Targeting mitochondrial calcium uptake with the natural flavonol kaempferol, to promote metabolism/secretion coupling in pancreatic β-cells
Nutrients, Vol. 12, Núm. 2
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Targeting mitochondrial ion channels in Type 2 diabetes
Future Medicinal Chemistry, Vol. 12, Núm. 17, pp. 1525-1527
2019
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Cellular stress is a prerequisite for glucose-induced mitochondrial matrix alkalinization in pancreatic β-cells
Molecular and Cellular Endocrinology, Vol. 481, pp. 71-83
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Regulation of inositol 1,4,5-trisphosphate-induced Ca2+ release from the endoplasmic reticulum by AMP-activated kinase modulators
Cell Calcium, Vol. 77, pp. 68-76
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The plant product quinic acid activates Ca2+-dependent mitochondrial function and promotes insulin secretion from pancreatic beta cells
British Journal of Pharmacology, Vol. 176, Núm. 17, pp. 3250-3263
2017
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A novel ATP-synthase-independent mechanism coupling mitochondrial activation to exocytosis in insulin-secreting cells
Journal of Cell Science, Vol. 130, Núm. 11, pp. 1929-1939
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Advances in pancreatic islet monolayer culture on glass surfaces enable super-resolution microscopy and insights into beta cell ciliogenesis and proliferation
Scientific Reports, Vol. 7
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Antibiotics induce mitonuclear protein imbalance but fail to inhibit respiration and nutrient activation in pancreatic β-cells
Experimental Cell Research, Vol. 357, Núm. 2, pp. 170-180